Myalgic Encephalomyelitis Blogger

Misdiagnosed Chronic Fatigue Syndrome (currently being mislabelled as ME/CFS when the IOM/NAM recommendation was to be changed to SEID) and the vagus nerve infection hypothesis The Vagus Nerve Infection Hypothesis (VNIH) proposes that, in some individuals, the symptoms of Chronic Fatigue Syndrome (CFS) are caused by an infection in or around the vagus nerve , the longest nerve of the autonomic nervous system in the human body. In 2013, Michael VanElzakker , then a graduate student at Tufts University and now a postdoctoral researcher at Harvard Medical School, published the hypothesis. The vagus nerve , also called the tenth cranial nerve, starts in the brain and runs down the trunk of the body, with branches that innervate all of the major organs. [ 2 ] It is responsible for the sickness response , an involuntary response characterized by fatigue , fever , myalgia , depression , and other symptoms that are often observed in patients with CFS . [ 3 ] Theory As explain...

ENTEROVIRUSES And Myalgic Encephalomyelitis (M.E.) INFECTION • IMMUNE RESPONSE • LASTING IMPACT Enteroviruses: Coxsackie B Echovirus Other numbered enteroviruses Enterovirus A71 Enterovirus D68 Coxsackie A BACKGROUND Patients "misdiagnosed" with Chronic Fatigue Syndrome (CFS) may have preceding illnesses, environmental exposures or co-existing conditions that warrant further investigation. Reported associations include sick building syndrome, mould exposure, prolonged stress, strenuous physical exertion, multiple chemical sensitivity and a history of silicone breast implants. Co-existing conditions such as Gilbert's syndrome, atopy, endometriosis, asthma, eczema and coeliac disease may also be relevant to the clinical assessment. Gluten sensitivity and associated neurological symptoms warrant consideration where clinically indicated. Episodes of epididymo-orchitis have also been reported preceding prolonged illness in male patients. ENTEROVIRUS...

Polio By Any Other Name? Knowing that there have been comparatively mild poliovirus outbreaks that damaged the brain and sometimes the spinal cord without causing paralysis or even weakness may dispose of some long-standing Polio Fictions, explain new findings about post-polio syndrome, and solve a mystery that’s nearly sixty years old. One Polio Fiction has already been banished: We know that “non-paralytic” polio is a misnomer. David Bodian showed in 1949, and Alan McComas confirmed in 1997, that paralytic and “non-paralytic” polio are not separate conditions but a single disease whose obvious symptoms are determined by the location and number of neurons killed by the poliovirus. Bodian even found that more brain stem neurons can be damaged, and damaged more extensively, in “non-paralytic” than in paralytic polio. As a matter of fact, the location of damaged brain neurons Bodian found in “non-paralytic” polio was the same as in monkeys injected w...